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Furthermore, the homozygous ApoE4-R136S mutation (ApoE4-Ch) was shown to rescue ApoE4-driven tau pathology, neurodegeneration, and neuroinflammation in tauopathy mouse models and human iPSC-derived neurons carrying human ApoE4 with either homozygous or heterozygous R136S mutations in late-onset AD

Weinlich R, Oberst A, Beere HM, Green DR

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Glisin (The Finisher) Glisin adalah potongan terakhir yang melengkapi struktur tripeptida ini