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Animal studies show some tissue repair, but reputable orthopedic organizations advise skepticism for human arthritis or injury
This improvement was associated with a modulation of the endoplasmic reticulum (ER) stress response, reflected in suppressed levels of C/EBP homologous transcription factor protein (CHOP), a proapoptotic factor, and increased expression of glucose regulatory protein 78, a chaperone protecting against ER stress
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Patients with chronic kidney diseases exhibit increased activity of xanthine oxidase (XO)
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